Background: A trigger to enhance Full Thickness Wound (FTW) repair is delivery of oxygen and
nutritients to the wound site. After the initial inflammatory phase, platelets and
keratinocytes (KC) start to secrete Vascular Endothelial Growth Factor (VEGF) to attract
Endothelial Cells into the wound granulation tissue. We present a method of ex vivo
expansion of autologous KC and lipid mediated gene transfer of a regulable VEGF165
plasmid for the treatment of FTW.
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© 2007 Published by Elsevier Inc.
