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Abstract
Cooling to 6–7° significantly increases ischaemic tissue survival to 48 and, probably, 72 hours. Inflammatory changes develop in direct proportion to the duration of the cold ischaemia time. The skin and vessels within the flap appear to be minimally affected by cold ischaemia while fat and muscle demonstrate significant necrosis, inflammation and ultimate fibrosis. Very little, if any, muscle survives the ischaemic insult studied in this model.
References
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Article info
Footnotes
This project was supported by a grant from the National Health and Medical Research Council of Australia.
Identification
Copyright
© 1980 The Trustees of British Association of Plastic Surgeons. All rights reserved. Published by Elsevier Inc.
